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Category Archives: Ventricular Remodeling

Ventricular hypertrophy – Wikipedia

Posted: November 19, 2018 at 8:40 am

Ventricular hypertrophy (VH) is thickening of the walls of a ventricle (lower chamber) of the heart.[1][2] Although left ventricular hypertrophy (LVH) is more common, right ventricular hypertrophy (RVH), as well as concurrent hypertrophy of both ventricles can also occur. Ventricular hypertrophy can result from a variety of conditions, both adaptive and maladaptive. Continue reading

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Ventricular dysfunction in heart failure – Britannica.com

Posted: at 8:40 am

Ventricular dysfunction in heart failure The major role of the ventricles in pumping blood to the lungs and body means that even a slight decrease in ventricular efficiency can have a significant impact on heart function. If the left ventricle encounters either absolute or relative functional insufficiency (called left ventricular heart failure, or left-sided heart failure), a series of compensatory reactions are initiated that may temporarily provide a return to sufficient ventricular function. Continue reading

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Ventricular hypertrophy – Wikipedia

Posted: November 18, 2018 at 1:46 am

Ventricular hypertrophy (VH) is thickening of the walls of a ventricle (lower chamber) of the heart.[1][2] Although left ventricular hypertrophy (LVH) is more common, right ventricular hypertrophy (RVH), as well as concurrent hypertrophy of both ventricles can also occur. Continue reading

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Ventricular dysfunction in heart failure – Britannica.com

Posted: at 1:46 am

Ventricular dysfunction in heart failure The major role of the ventricles in pumping blood to the lungs and body means that even a slight decrease in ventricular efficiency can have a significant impact on heart function. If the left ventricle encounters either absolute or relative functional insufficiency (called left ventricular heart failure, or left-sided heart failure), a series of compensatory reactions are initiated that may temporarily provide a return to sufficient ventricular function Continue reading

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Ventricular remodeling – Wikipedia

Posted: November 16, 2018 at 2:41 pm

In cardiology, ventricular remodeling (or cardiac remodeling)[1] refers to changes in the size, shape, structure, and function of the heart. This can happen as a result of exercise (physiological remodeling) or after injury to the heart muscle (pathological remodeling).[2] The injury is typically due to acute myocardial infarction (usually transmural or ST segment elevation infarction), but may be from a number of causes that result in increased pressure or volume, causing pressure overload or volume overload (forms of strain) on the heart Continue reading

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Ventricular Remodeling Surgery (SVR)

Posted: September 24, 2018 at 5:45 am

Surgical Ventricular Reconstruction (SVR) Surgical ventricular reconstruction (SVR) reduces the size of a patient’s failing, enlarged heart to ensure that blood is pumped out of the heart, and through the body efficiently enough to allow them to participate in a full, active life. In a healthy patient, oxygen-rich blood returning from the lungs is pumped through the heart and to the body. Continue reading

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Left ventricular hypertrophy – Wikipedia

Posted: August 20, 2018 at 12:43 pm

Left ventricular hypertrophy (LVH) is thickening of the heart muscle of the left ventricle of the heart, that is, left-sided ventricular hypertrophy. While ventricular hypertrophy occurs naturally as a reaction to aerobic exercise and strength training, it is most frequently referred to as a pathological reaction to cardiovascular disease, or high blood pressure.[1] It is one aspect of ventricular remodeling. While LVH itself is not a disease, it is usually a marker for disease involving the heart.[2] Disease processes that can cause LVH include any disease that increases the afterload that the heart has to contract against, and some primary diseases of the muscle of the heart. Continue reading

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Ventricular Remodeling | Profiles RNS

Posted: July 24, 2018 at 11:45 pm

“Ventricular Remodeling” is a descriptor in the National Library of Medicine’s controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity. The geometric and structural changes that the HEART VENTRICLES undergo, usually following MYOCARDIAL INFARCTION. Continue reading

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Acute Management of Pulmonary Embolism – American College …

Posted: July 17, 2018 at 2:45 am

Introduction Venous thromboembolic disease (VTE) is estimated to occur in at least 1 to 2 persons per 1000 population annually, manifesting as deep vein thrombosis (DVT), pulmonary embolism (PE) or in combination.1-3 It is the cause of over 100,000 deaths annually and is the most preventable cause of death in hospitalized patients in the United States.4 Despite treatment with anticoagulant therapy, a significant proportion of survivors of acute DVT or PE are at risk of suffering from the disabling sequelae such as the post thrombotic syndrome (PTS), recurrent VTE or chronic thromboembolic pulmonary hypertension (CTEPH).1,5 Given the limitations of medical therapy, promising endovascular treatment modalities have evolved over the past two decades in an effort to mitigate the acute and chronic disability from VTE.6,7 The purpose of this review is to discuss the rationale and evidence for an endovascular treatment approach for high-risk acute DVT and PE patients. The Rationale for an Interventional Approach to Massive and Submassive PE The most dreaded acute complication of PE is death; it is estimated that over 100,000 deaths in hospitalized patients in the United States are attributable to acute PE each year.4 The severity of PE is stratified into massive (PE causing hemodynamic compromise), submassive (PE causing right ventricular dysfunction demonstrable by echocardiography, computed tomography or elevated cardiac biomarkers) and non-massive or low-risk (PE without evidence of RV dysfunction or hemodynamic compromise). The International Cooperative Pulmonary Embolism Registry (ICOPER) demonstrated 90-day mortality rates of 58.3% in patients with massive PE versus 15.1% in sub-massive PE.20 Several studies demonstrate short-term mortality rates of less than 2% in patients with low-risk PE.21-23 Features suggestive of adverse prognosis in acute PE are listed in Table 2. Continue reading

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Heart Failure Medication: Beta-Blockers, Alpha Activity …

Posted: July 7, 2018 at 10:42 am

Ho KK, Pinsky JL, Kannel WB, Levy D. The epidemiology of heart failure: the Framingham Study. Continue reading

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