Cancer DNA Sequenced Professor Sean Grimmond's team from the Queensland Centre for Medical Genomics, based at UQ Institute for Molecular Biosciences , has spent five years working closely with Sydney's Garvan... By: The University of Queensland … Continue reading →

Marsha Berry Toronto Real Estate 1030 King St W. DNA 3 487SF 1 Bedroom This video is about Marsha Berry Toronto Real Estate 1030 King St W. 494SF 1 Bedroom. By: Marsha Berry CondosThroughYourEyes … Continue reading →

Higher Ascension - DNA (live at Modesto X-Fest) Higher Ascension performing DNA live at X-Fest 2014 in Modesto, Ca. By: Enrique Burgos … Continue reading →

A team of scientists led by Research Associate Professor Motomi Osato and Professor Yoshiaki Ito from the Cancer Science Institute of Singapore (CSI Singapore) at the National University of Singapore (NUS) found that a drug originally designed for killing a limited type of cancer cells with DNA repair defects could potentially be used to treat leukemia and other cancers. The new study suggests that treatment with poly (ADP-ribose) polymerase (PARP) inhibitors, together with standard chemotherapy drugs, could be more effective in combating leukemia. In the same study, researchers found that the inactivation of RUNX genes causes DNA repair defects and promotes the development of leukemia and other cancers. The study was first published online in the leading journal Cell Reports last month. Unlike other cancers which are more commonly seen in the elderly, leukemia is notorious for its high prevalence among young people. There has been little advancement in the treatment of leukemia. Chemotherapy with or without hematopoietic stem cell transplantation remains the current standard of care, resulting in a cure rate of around 50 per cent. The RUNX family genes are among the most frequently inactivated genes in leukemia and other cancers. According to previous studies, RUNX1 is one … Continue reading →

Arturo Valtierra-Payan, center, listens to the first day of testimonay Monday in the 243rd District Court. Valtierra-Payan is charged in a series of sexual assaults that occurred in the Manhattan Heights neighborhood last year. (RUBEN R RAMIREZ EL PASO TIMES) Replay Twitter updates: Trial for accused Manhattan Heights predator DNA evidence connecting a man charged with a series of sexual assaults last year in the Manhattan Heights neighborhood to one of the victims was presented Monday at the start of his trial. Arturo Valtierra-Payan's trial began Monday in the 243rd District Court. Valtierra-Payan, 26, is charged with three counts of aggravated sexual assault and one of count each of burglary of habitation and aggravated robbery in the case. Prosecutors opened the trial detailing the night of June 26, 2013 when Valtierra-Payan allegedly broke into a home in the Manhattan Heights neighborhood. According to an opening statement by state prosecutor Penny Hamilton and testimony from law enforcement officers, Valtierra-Payan allegedly attempted to get into the home through a window at about 1 a.m. Valtierra-Payan then allegedly broken down the front door and attempted to rape the woman who lived in the home, Hamilton told jurors. According to multiple law enforcement officers … Continue reading →

PUBLIC RELEASE DATE: 25-Aug-2014 Contact: Shawna Williams shawna@jhmi.edu 410-955-8236 Johns Hopkins Medicine Regardless of their stage or type, cancers appear to share a telltale signature of widespread changes to the so-called epigenome, according to a team of researchers. In a study published online in Genome Medicine on Aug. 26, the investigators say they have found widespread and distinctive changes in a broad variety of cancers to chemical marks known as methyl groups attached to DNA, which help govern whether genes are turned "on" or "off," and ultimately how the cell behaves. Such reversible chemical marks on DNA are known as epigenetic, and together they make up the epigenome. "Regardless of the type of solid tumor, the pattern of methylation is much different on the genomes of cancerous cells than in healthy cells," says Andrew Feinberg, M.D., M.P.H., a professor of medicine, molecular biology and genetics, oncology, and biostatistics at the Johns Hopkins University School of Medicine. Feinberg led the new study along with Rafael Irizarry, Ph.D., a professor of biostatics at Harvard University and the Dana-Farber Cancer Institute. "These changes happen very early in tumor formation, and we think they enable tumor cells to adapt to changes in their environment … Continue reading →